1. Phellodendrine has anti-nephritic activity, may be due to its ability to inhibit the proliferation or the migration of macrophages and cytotoxic T lymphocytes in the glomeruli.
1. Coenzyme Q10 is used in the treatment of Huntington's disease.
2. Coenzyme Q10 Inhibits the Aging of Mesenchymal Stem Cells Induced by D-Galactose through Akt/mTOR Signaling.
3. Coenzyme Q10 is a cardiovascular agent, used to treat congestive heart failure; can improve energy production in cells, prevent blood clot formation, and act as an antioxidant.
1. Astragaloside IV has antioxidant and anti-aging activities.
2. Astragaloside IV protects dopaminergic neurons.
3. Astragaloside IV has anti-fibrotic effect against systemic sclerosis.
4. Astragaloside IV has anti-inflammatory activity, the mechanism may be inhibiting the NF-kappaB pathway.
5. Astragaloside IV inhibits migration and invasion in human lung cancer A549 cells via regulating PKC-α-ERK1/2-NF-κB pathway.
1. Dioscin has anticancer and anti-inflammatory activity.
2. Dioscin is an immunomodulator, and an anticarcinomic agent.
3. Dioscin has antifungal activity, promotes healthy intestine Flora/Oiges live system.
4. Dioscin inhibits adipogenesis through the AMPK/MAPK pathway in 3T3-L1 cells.
5. Dioscin is considered a clinically useful GH alternative that induces GH release by pituitary stimulation, supports healthy bladder function.
6. Dioscin has antineoplastic activity, inhibit the growth of leukemia cells and inhibits colon tumor growth and tumor angiogenesis through regulating VEGFR2 and AKT/MAPK signaling pathways.
1. Mulberroside has nephroprotective, hypoglycemic, and antidiabetic effects.
2. Mulberroside A is a glycosylated stilbene of oxyresveratrol; thus, the deglycosylation of Mulberroside A resulted in enhanced inhibition of melanogenesis.
3. Mulberroside A has anti-inflammatory antiapoptotic effects. by decreasing the expressions of tumor necrosis factor-α (TNF-α), interleukin (IL)-1β, and IL-6 and inhibiting the activation of NALP3, caspase-1, and nuclear factor-κB and the phosphorylation of extracellular signal-regulated protein kinases, the c-Jun N-terminal kinase, and p38.