Hot Products
| Catalog No. | Information |
| CFN99724 | 3-Isomangostin 3-Isomangostin is a potent human aldose reductase inhibitor with an IC50 of 3.48 uM; it is also an acetylcholinesterase selective inhibitor. 3-Isomangostin has free radical scavenging activity; it shows antiplasmodial activity with IC50 values in the range of 4.71-11.40 uM. |
| CFN96186 | 3'-Methoxydaidzein 3'-Methoxydaidzein has antioxidant activity, it and geraldol have antiplatelet aggregation activity and appear to be specific against collagen‐induce platelet aggregation with a IC50 values of12.3 and 61.5 uM, respectively. |
| CFN89443 | 3'-Methoxydaidzin 3'-Methoxydaidzin has antioxidation activity, it may be a potential neuroprotective substance in Gegen Qinlian decoction. |
| CFN95112 | 3'-Methoxymirificin Reference standards. |
| CFN90780 | 3'-Methoxypuerarin 3'-Methoxypuerarin shows neuron protection activity, it can protect hippocampal neurons against ischemia/reperfusion injury by inhibiting apoptosis. 3'-Methoxypuerarin has antioxidant activities, it shows ONOO(-) scavenging activity and weak NO· and O(2)(-) scavenging activities. |
| CFN70390 | 3-Methyl ellagic acid 3-Methyl ellagic acid has anti-oxidant activity. |
| CFN97777 | 3-Methyl-9H-carbazol-2-ol Reference standards. |
| CFN60037 | 3-Methyladenine 3-Methyladenine (3-MA) is a selective PI3K inhibitor for Vps34 and PI3Kγ with IC50 of 25 μM and 60 μM in HeLa cells; blocks class I PI3K consistently, whereas suppression of class III PI3K is transient, and also blocks autophagosome formation. 3-Methyladenine (3-MA) is successfully used to suppress mitophagy. |
| CFN70209 | 3-Methylindole 3-Methylindole , a selective pneumotoxin, causes acute pulmonary edema and emphysema. |
| CFN90235 | 3-n-Butylphthalide Dl-3-n-Butylphthalide has antihypertensive effects, may slow the progression of hypertensive nephropathy by a variety of mechanisms.3-n-Butylphthalide is effective for improving cognitive and global functioning in patients with subcortical VCIND and exhibits good safety, this effect might be mediated by preventing the decline of the central cholinergic system. |
