Bioactive Products
A unique collection of 267 Inhibitor natural compounds | ||
Catalog No: | Bb221 | Inhibitors Compound Library Screening Details |
Size: | 1mg/well * 267 Compounds 2mg/well * 267 Compounds |
Cat. No. | Information |
CFN99959 | Huperzine A Huperzine A is a potent, selective and reversible acetylcholinesterase (AChE) inhibitor and has been widely used in China for the treatment of Alzheimer's disease (AD).Huperzine A induces CYP3A4 expression and activation via PXR dependent pathways, may contribute to drug-drug interactions with ligustrazine and oridonin. |
CFN99960 | Huperzine B Huperzine B is a efficient inhibitor of human brain AChE, it can enhance ognitive and protect neuro, may be potentially new drug candidates for Alzheimer's disease therapy. |
CFN98037 | Coniferaldehyde Coniferaldehyde exerts anti-inflammatory properties by inducing heme oxygenase-1(HO-1), it inhibits LPS-induced apoptosis through the PKC α/β II/Nrf-2/HO-1 dependent pathway in RAW264.7 macrophage cells. Coniferaldehyde can significantly inhibit the growth of viability of strains of Oenococcus oeni. |
CFN98038 | Sinapaldehyde Sinapaldehyde has effects against 65 strains of Candida,Sinapaldehyde inhibits prostaglandin synthetase in a dose-dependent way, it dose-dependently inhibits ethyl phenylpropiolate-induced edema of the rat ear, and can inhibit electrically induced contractions of the guinea pig ileum in a dose-dependent way.The reaction of sinapaldehyde and methylpyranoanthocyanin can obtain a new bluish pigment. |
CFN99968 | Ginsenoside Rg2 Ginsenoside Rg2 has therapeutic potential for type 2 diabetic patients, it also may represent a potential neurorestorative treatment strategy for vascular dementia or other ischemic insults, has protective effects against H2O2-induced injury and apoptosis in H9c2 cells. Ginsenoside Rg2 suppresses the hepatic glucose production via AMPK-induced phosphorylation of GSK3β and induction of SHP gene expression, regulates the 5-HT3A receptors that are expressed in Xenopus oocytes, inhibits nicotinic acetylcholine receptor-mediated Na+ influx and channel activity. |