Hot Products
| Catalog No. | Information |
| CFN98601 | Vitexin Vitexin, an HIF-1alpha inhibitor, which has anticonvulsant, anti-depressant, anti-glycation, spasmolytic, anti-metastatic, antitumor, anti-inflammatory and antinociceptive activities. Vitexin can be effectively used for the prevention of UV-induced adverse skin reactions such as free radical production and skin cell damage; it non-competitively inhibits Ach but not the Ca(2+) influx. |
| CFN70421 | Vitexin 2''-glucoside Reference standards. |
| CFN96447 | Vitexin 2''-O-(4'''-O-acetyl)rhamnoside Reference standards. |
| CFN90654 | Vitexin 2''-O-p-coumarate Vitexin 2''-O-p-coumarate strongly promotes 2BS cell proliferation induced by H(2)O(2). |
| CFN92072 | Vitexin -4''-O-glucoside Vitexin-4''-O-glucoside could effectively protect ECV-304 cells against cytotoxicity induced by TBHP through resuming mitochondrial function. |
| CFN70343 | Vitexin 7-glucoside Vitexin-7-glucoside exhibits high intestinal permeability, and predictive of excellent human absorption, which awaits confirmation from further investigation in vivo. |
| CFN98177 | Vitexin-2''-O-rhamnoside Vitexin-2''-O-rhamnoside contributes to the protection against H₂O₂ -mediated oxidative stress damage and could be safely used for a wide range of concentrations.It has low bioavailability, mainly related to its poor absorption in the intestine. |
| CFN92252 | Voacangine Voacangine is a novel transient receptor potential vanilloid type 1 (TRPV1) antagonist, it shows mod. cytotoxic activity, also some CNS, brachycardial and hypotensive action.Voacangine significantly suppresses in vitro angiogenesis, such as VEGF-induced tube formation and chemoinvasion. |
| CFN96444 | Vogeloside Vogeloside shows inhibition of nitric oxide production in lipopolysaccharide-activated macrophages; its activity is comparable to that of aminoguanidine, a classic inhibitor. |
| CFN97192 | Voleneol 1β,6α-Dihydroxyeudesm-4(15)-ene (Voleneol,DE) can attenuate the lipopolysaccharide (LPS)-induced inflammation in BV2 microglial cells, it also can dose-dependently suppress the protein expression of inducible nitric oxide synthase (iNOS) and cyclooxygenase-2 (COX-2),suggest thats DE may be a good candidate to regulate LPS-induced inflammatory response. |
