Hot Products
| Catalog No. | Information |
| CFN93061 | 2-Pentylfuran 2-Pentylfuran is a naturally occuring flavouring ingredient, it may be a biomarker for lung colonization/infection by fungal pathogens. 2-Pentylfuran and isomers of 2-pentenylfuran are known to be mainly responsible for the undesirable reversion flavor of soybean oil. |
| CFN00048 | 2S-Amino-3R-octadecanol 2S-Amino-3R-octadecanol inhibits cell proliferation,it is an antitumour agent. |
| CFN99022 | 3-(Hydroxymethyl)cyclopentanol Reference standards. |
| CFN98299 | 3,19-Dihydroxy-6,23-dioxo-12-ursen-28-oic acid Reference standards. |
| CFN90576 | 3,29-Dibenzoyl karounitriol 3,29-Dibenzoyl karounitriol is a natural product from Trichosanthes kirilowii Maxim. |
| CFN91175 | 3,29-O-Dibenzoyloxykarounidiol |
| CFN95021 | 3,3',4',5,6,7,8-heptamethoxyflavone 3,3',4',5,6,7,8-Heptamethoxyflavone(HMF) has neuroprotective, chemopreventive, anti-allergic, anti-inflammatory, immunomodulatory, anti-aging and photoprotective effects; it inhibits collagenase activity and induces type I procollagen synthesis in HDFn cells, and against nitric oxide (NO) carcinogenesis. |
| CFN90830 | 3,3'-Di-O-methylellagic acid 4'-glucoside Reference standards. |
| CFN70332 | 3',4',5,5',6,7-Hexamethoxyflavone 5,6,7,3',4',5'-Hexamethoxyflavone(HMF) has anticancer activities, it inhibits growth of triple-negative breast cancer cells via suppression of MAPK and Akt signaling pathways and arresting cell cycle. HMF may potentiate Cystic fibrosis transmembrane conductance regulator (CFTR) Cl(-) channel activities through both elevation of cAMP level and binding to CFTR protein pathways, HMF may be developed as a new drug in the therapy of CFTR-related diseases such as bronchiectasis and habitual constipation. |
| CFN90238 | 3,4,5-Tricaffeoylquinic acid 3,4,5-Tricaffeoylquinic acid may attenuate the TNF-α- and LPS-stimulated production of inflammatory mediators in keratinocytes by suppressing the Toll-like receptor 4 expression-mediated activation of the Akt, ERK and NF-ĸB pathways, it may exert an inhibitory effect against the pro-inflammatory mediator-induced skin disease. 3,4,5-Tricaffeoylquinic acid may attenuate the proteasome inhibitor-induced programmed cell death in PC12 cells by suppressing the activation of the mitochondrial pathway and the caspase-8- and Bid-dependent pathways, the effect be attributed to its inhibitory effect on the formation of reactive oxygen species and depletion of GSH. |
